Back and Joint Pain-New Relief Through Lipoxygenase Inhibition With Natural Supplements

Lipoxygenase Inhibition: A Missed opportunity forIf the label does not specifically state that the AKBA
controlling pain and inflammationcontent is minimum 90% you are not getting the
Are we only "half treating" our back and joint pains?best quality possible.
Back and joint pains are among the top reasons forWith the right quality and dosage, however, either
visits to the doctor. Yet complete resolution of thetaken alone or together with other therapies
complaint is often slow in coming or never completelysymptom relief could be seen from this nutritional
resolved. Why?modality. Improvement in previously therapy resistant
There are 2 major physiologic pathways leading toback or joint pain, prolonged "holding" of chiropractic
inflammatory and pain responses: the cyclooxygenaseadjustments and faster pain relief after injury has
(COX) mediated and the lipoxygenase (5-LO)been routinely noted.
dependent one. While the former is widely known,Safety and toxicology;
and inhibited by the well popularized NSAID's (likeHigh concentration boswellia extracts are considered
Celebrex, Vioxx(!)), etc, the second one is virtuallyGRAS - generally regarded as safe. There are
ignored in the current management of painpractically no side effects except for an occasional
syndromes. Principal cause for this is the failure ofreport of headache. There have not been any
major drug companies to develop synthetic drugsreports of the intestinal distress seen with other
that can inhibit the 5-LO and its downstreamboswellia preparations.
metabolites, the all important leukotrienes. To makeConclusion
up for the failure to deal with the 5-LO,High concentration boswellia extracts with 90% or
pharmaceutical solutions like corticosteroids are usuallymore AKBA is highly beneficial for the treatment of
put in place - with their well known side effects.pain syndromes ranging from back to joints and
The bottom line is that when only one of twoother damaged soft tissues. They can help in other
pathways to pain is addressed the patient is onlyorgan and neurological pain conditions due to their
"half treated" and half satisfied.anti- inflammatory properties. It can be given as a
Lipoxygenase and painstand alone solution or in conjunction with other COX
The 5-LO enzyme works to produce the "misery" ofinhibitors. They are considered nutritional supplements
the leukotrienes.1 They are abundantly involved inand should be part of a general health maintenance.
over 35 chronic conditions including: asthma, allergies,(These statements have not been evaluated by the
colitis, arthritis, gastric disorders (promote ulcerFDA. These ingredients are not intended to diagnose,
formation, stimulate acid secretion, etc), scleroderma,treat, cure, or prevent any disease. Never start a
neurological diseases, and so on. 2new program without consulting a qualified heath care
More recently the involvement of the leukotrienes inprofessional.)
pain syndromes has become clear from a multitudeReferences
of studies. 11. Whitehouse MW, Rainsford KD. Lipoxygenase
5-LO and leukotriene B4 are involved in orofacial paininhibition: The neglected frontier for regulating chronic
perception and mechanical and thermal sensitivity.3-7inflammation and pain. Inflammopharmacology.
Postoperative incision pain in animal models could be2006;14( 3-4):99-102. 2. Werz O, Steinhilber D.
considerably reduced using experimental 5-LOPharmacological intervention with 5-lipoxygenase: New
inhibitors.8insights and novel compounds. Expert Opinion on
The beneficial effects of 5-LO inhibition wereTherapeutic Patents. 2005;15( 5):505-519. 3. Aley KO,
demonstrated in the reduction of inflammatoryLevine JD. Contribution of 5- and 12-lipoxygenase
events accompanying experimental spinal cord injury.products to mechanical hyperalgesia induced by
9prostaglandin E2 and epinephrine in the rat.
Several studies have identified inflammatoryExperimental Brain Research. 2003;148( 4):482-487. 4.
mediators in disk herniation, such as leukotrienes.Amann R, Schuligoi R, Lanz I, Peskar BA. Effect of a
Cytokines occurring in degenerated facets have been5-lipoxygenase inhibitor on nerve growth
shown to contribute to the pain of degenerativefactor-induced thermal hyperalgesia in the rat.
lumbar disorders. 10, 11European Journal of Pharmacology. 1996;306(
5-LO has been shown to be involved in both pain1-3):89-91. 5. Bisgaard H, Kristensen JK. Leukotriene
modulation and induction of opioid tolerance at theB4 produces hyperalgesia in humans. Prostaglandins.
spinal level. 12 5-LO metabolites are found in clinical1985;30( 5):791-797. 6. Chichorro JG, Lorenzetti BB,
cases of herniated nucleus pulposus and experimentalZampronio AR. Involvement of bradykinin, cytokines,
data gathered in the study of associated radicularsympathetic amines and prostaglandins in
pain in animals demonstrated that 5-LO inhibition mayformalin-induced orofacial nociception in rats. British
prove to be beneficial in such conditions.13Journal of Pharmacology. 2004;141( 7):1175-1184. 7.
Pharmacological inhibition of the 5-LOMartin HA. Leukotriene B4 induced decrease in
While the market availability of COX inhibitors ismechanical and thermal thresholds of C-fiber
widespread the opposite seems to be the case withmechanonociceptors in rat hairy skin. Brain Research.
pharmaceuticals in the lipoxygenase class direction.1990;509( 2):273-279. 8. Gaspar AF, Prado WA.
Partially this is not due to lack of trying. PromisingComparison of pre- versus post-incision administration
experimental drugs had to be abandoned due toof intraplantar indomethacin and MK886 in a rat model
unacceptable side effects - death of animal subjects!of postoperative pain. Brazilian Journal of Medical and
Even those that made it to market carry warnings ofBiological Research. 2007;40( 8):1141-1147. 9.
hepatotoxicity (Zileuton) or have been associatedGenovese T, Rossi A, Mazzon E, et al. Effects of
with an increase in abnormal mental behaviorzileuton and montelukast in mouse experimental spinal
(Singulair). On the other hand there is a persistentcord injury. British Journal of Pharmacology. 2008;153(
lack of research on the part of the pharmaceutical3):568-582. 10. Goupille P, Jayson MIV, Valat J-,
industry secondary to a tragic underestimation of theFreemont AJ. The role of inflammation in disk
potential market size.herniation-associated radiculopathy. Seminars in
Financial disincentives explain the lack of studies ofArthritis and Rheumatism. 1998;28( 1):60-71. 11.
extracts of "natural" substances which are not easilyIgarashi A, Kikuchi S, Konno S, Olmarker K.
patentable.Inflammatory cytokines released from the facet joint
Advances in nutritional therapy with hightissue in degenerative lumbar spinal disorders. Spine.
concentration boswellia (frankincense) extracts2004;29( 19):2091-2095. 12. Trang T, McNaull B,
The premier 5-LO inhibitor is the natural, herbalQuirion R, Jhamandas K. Involvement of spinal
ingredient AKBA, acetyl-11-keto-beta-boswellia acid,lipoxygenase metabolites in hyperalgesia and opioid
the most active component of the frankincense,tolerance. European Journal of Pharmacology.
Boswellia serrata. Boswellia as such has been known2004;491( 1):21-30. 13. Singh VP, Patil CS, Kulkarni SK.
for centuries to be a potent anti-inflammatory agent.Effect of licofelone against mechanical hyperalgesia
Studies have proven its efficacy in arthritis, colitis,and cold allodynia in the rat model of incisional pain.
allergies and environmental sensitivities. 14-16Pharmacological Reports. 2005;57( 3):380-384. 14.
More recent studies have confirmed the analgesicAmmon HPT. Boswellic acids in chronic inflammatory
properties of boswellia extracts, both as stand alonediseases. Planta Medica. 2006;72( 12):1100-1116. 15.
solutions as well as synergistic enhancers of pain reliefAmmon HPT. Boswellic acids for the treatment of
when given in conjunction with COX inhibitors, opioidschronic inflammatory diseases. Medizinische
and other NSAID's. 17, 18Monatsschrift fur Pharmazeuten. 2003;26( 9):309-315.
The success of boswellia extracts is all the more16. Poeckel D, Werz O. Boswellic acids: Biological
surprising since only poorly standardized productsactions and molecular targets. Current Medicinal
have been available on the general market. TheChemistry. 2006;13( 28):3359-3369. 17. Bishnoi M, Patil
component AKBA is recognized as the active anti-CS, Kumar A, Kulkarni SK. Analgesic activity of
inflammatory principle in the boswellia and yet by faracetyl-11-keto-beta-boswellic acid, a
most of the formulas have only 1-3% AKBA5-lipoxygenase-enzyme inhibitor. Indian Journal of
concentration.Pharmacology. 2005;37( 4):255-256. 18. Bishnoi M, Patil
Fortunately high concentration boswellia extracts areCS, Kumar A, Kulkarni SK. Protective effects of
now available with a concentration of over 90%nimesulide (COX inhibitor), AKBA (5-LOX inhibitor), and
AKBA! This leads to enhanced efficacy. There are atheir combination in aging-associated abnormalities in
number of high quality boswellia products on themice. Methods and Findings in Experimental and Clinical
market. To get the best a careful reading of thePharmacology. 2005;27( 7):465-470.
supplement facts on the label is necessary.