Glutathione - Your Brain's Master Antioxidant Defense

Free radicals and oxyradicals play an important role inStudies have found that the mood stabilizing drug,
the development and progression of many brainvalproate, used to treat epilepsy and bi-polar disorder,
disorders such as brain injury, neurodegenerativeregulates expression of the genes that make
disease, schizophrenia and Down syndrome.glutathione-S-transferase (GST).
Glutathione is the brain's master antioxidant and playsIn addition, chronic treatment with lithium, another
an important protective role in the brain.commonly prescribed mood stabilizer used in treating
According to Dr. Jimmy Gutman, "The brain ismanic-depression, also increased levels of GST.
particularly susceptible to free radical attack becauseThese findings led researchers to conclude that
it generates more oxidative by-products per gram ofglutathione S-transferase may be a novel target for
tissue than any other organ."mood stabilizing drugs. (7)
Many neurological and psychiatric disease processesAlcohol Consumption and Glutathione
are characterized by... abnormalities in glutathioneAlcohol abuse is known to impair memory and other
metabolism and antioxidant defenses."brain functions and increase brain cell death. A new
Generation of reactive oxygen species (free radicals)study in rats has shown that alchol consumption
and oxidative damage are an important cause ofcauses fewer new brain cells to form and results in
neuron (brain cell) death from brain injury.greater cell death. (8)
Chemicals that cause toxicity to certain brain cells areBut rats that were fed alcohol along with Ebselen - a
known to decrease cerebral glutathione (GSH),glutathione peroxidase mimic that acts as a free
making the cells more vulnerable to reactive oxygenradical scavenger - showed no similar reduction in
species (ROS). (1)brain-cell formation and no increase in cell death.
On the other hand, over-expression of theSubstances that Boost Glutathione Levels and
glutathione peroxidase (GPX) enzyme potentlyProtect Brain Cells
decreases cell death from brain injury. (2)Taking glutathione itself as a supplement does not
Brain Injury and Glutathione - The Gender Differenceboost cellular glutathione levels, since it breaks down
Researchers at Children's Hospital of Pittsburgh havein the digestive tract before it reaches the cells.
found that males and females respond differently toHowever, intravenous glutathione therapy and
brain injury. (3)glutathione precursors or dietary supplements are
In animal models, levels of glutathione remaineffective in boosting intracellular levels of glutathione.
constant in females who have suffered a brain injury,Intravenous Glutathione Injections: Intravenous
but drop by as much as 80 percent in males with theglutathione injections have been shown to produce
same injury.amazing and rapid results, in patients with Parkinson's
When glutathione levels drop, brain cells die muchdisease. Following even a single dosage of
more quickly. This suggests that boys with brainintravenous glutathione, many of the symptoms of
injuries may require different life-saving treatmentsParkinson's disease rapidly improve, often in as little
than girls.as 15 minutes.
N-acetyl-cysteine (NAC), a precursor of glutathione,Glutathione Precursors: In the Alzheimer's study
already approved for use by the U.S. Food and Drugconducted by Welsh GP, Andrew McCaddon, adding
Administration to treat people who have overdosedthe glutathione precursor, N-acetyl-cysteine (NAC) to
on acetaminophen, may be an effective treatmenta protocol that lowered homocysteine levels by
for brain injury in boys whose brains are deprived ofsimple supplementation with B12 and folate, resulted
oxygen.in prompt, striking, and sustained clinical improvement
Brain Disorders and Glutathione - A Genetic Cause?in nearly all the patients. (9)
Genetics researchers have found that the glutathioneCucurmin (turmeric):Studies have shown that the
S-transferase gene controls the onset of Alzheimer's,Indian curry spice, cucurmin, has neuroprotective
Parkinson's disease and determines, not if we geteffects because of its ability to induce the enzyme,
these diseases, but when. (4)hemeoxygenase-1 (HO-1), which protects neurons
The glutathione S-transferase gene has previouslyexposed to oxidant stress. Treatment of brain cells
been linked to the risk for Parkinson's disease amongcalled astrocytes, with curcumin, increases expression
people who used pesticides.of HO-1 protein as well as glutathione S-transferase.
A previous article covered the importance of(10)
glutathione in Parkinson's Disease.Ebselen: Ebselen is a glutathione peroxidase mimic and
Alzheimer's Disease and Glutathionepotent synthetic antioxidant that acts as a
Free radicals and oxidative damage in neurons isneuroprotective agent and an inhibitor of free-radical
known to be a primary cause of degenerativeinduced apoptosis (cell death). It can protect brain
diseases like Alzheimer's disease.cells from the neuro-toxic effects of alcohol
Amyloid-ß peptide (Aß) accumulation inconsumption. (8)
senile plaques, a pathological hallmark of Alzheimer'sUndenatured Whey Protein: Undenatured whey
disease (AD), has been implicated in neuronalprotein provides glutathione precursors, has been
degeneration.shown to raise intracellular glutathione levels in clinical
Amyloid plaques encroaching on the brain increase thetrials, and has anecdotally been reported to improve
production of free radicals, or oxidative stress.the symptoms of Parkinson's disease.
Antioxidants, such as vitamin C and E "mop up" theReferences:
damaging free radicals.1. Journal of Neurochemistry, Vol. 88, No. 3, 2004
Glutathione (GSH) precursors can prevent death of513-531
brain cells induced by amyloid plaques in Alzhiemer's2. Journal of Neurochemistry, Vol. 87, No. 6, 2003
disease, while substances that deplete GSH increase1527-1534
cell death. (5)3. Researchers Find Brain Cells Die Differently in Males
Evidence has been piling up over the link betweenand Females; Pediatric Academic Societies Press
the amount of an amino acid called homocysteine inrelease; 21-Apr-2004
the blood and the chance of developing Alzheimer's.4. Human Molecular Genetics, 2003, Vol. 12, No. 24
For people not genetically predisposed to developing3259-3267
Alzheimer's, cholesterol and homocysteine, largely5. The Journal of Cell Biology, Volume 164, Number 1,
caused by an unhealthy lifestyle, are the core causal123-131; 5 January 2004
factors.6. Biol Psychiatry. 2003 Feb;53(3):254-60
Welsh GP, Andrew McCaddon, showed that the7. Journal of Neurochemistry, Vol. 88, No. 6, 2004
more homocysteine that patients with Alzheimer's1477-1484
had, the worse their mental performance, and the8. Proc Natl Acad Sci U S A. 2003 Jun
worse their "cognitive impairment," the less they had24;100(13):7919-24. Epub 2003 Jun 05.
of the antioxidant glutathione. (6)9. Am J Geriatr Psychiatry. 2003 Mar-Apr;11(2):246-9
Glutathione and Mood Disorders10. Can Curry Protect Against Alzheimer's?