Living With ALS

Hello, my name is Buddy Sowell and I have ALS (Loucontrolled self-renewal is needed. Neuronal progenitor
Gehrig's Disease). In September 2007 I visited acells have also been shown to persist into adulthood
Neurologist to get some answers as to why myin specific brain locations near the ventricles where
muscles were persistently twitching. I had somethey support ongoing learning and the establishment
muscle cramping & slight fatigue, but thoughtof new memories through their division,
nothing of it. After some seemingly useless muscledifferentiation, migration, and insertion into new
strength testing, Dr Dooley looked at me withcircuitry.
obvious concern on his face. I thought it was maybeIs There a Role for Stem Cell Therapy? Stem cells
the fact that he was in his upper 80s and he alwayscould help patients with ALS in several ways. Ideally,
looked that way...anyway, he suggested that I see athey could be induced to differentiate into lower
Neurology specialist named Dr. Robert Baloh atmotor neurons in order to replace those neurons that
Washington University Medical Center. After somedie because of ALS. Perhaps stem cells could rescue
intense and painful testing and months of excruciatingdying motor neurons by reconnecting these neurons
waiting... I was surprisingly diagnosed with ALS.to partly denervated muscle before it has died
"You've got to be kidding" I remember saying...completely. Better yet, they could be induced to
Never in a million years would I have guessed thatdifferentiate into upper motor neurons in the cortex
something like this would have happened to me andand connect to the lower motor neurons.
to my family. My wife Lori and my daughters CarlyUnfortunately, the expectation that stem cells will
and Casey are my entire world. I love them moreplay such a regenerative role in patients with Lou
than words can describe. They have all made me aGehrig's disease is unrealistic because of the
better person. My prognosis is uncertain, but I willcomplexity of the task, which presents obstacles
fight. My loving family doesn't deserve this...so I fightthat currently are insurmountable. A more realistic
for them.expectation for stem cells is that they play a
Lori and I talked canididly about what we should dosupportive role in maintaining the viability of or
next. We decided to take advantage of what timeextending the function of surviving motor neurons.
we still have together and focus on having fun as aThe stem cells could be induced to differentiate into
family. No more putting off travel plans. Let's try tosupporting cells, glia, or interneurons that might
do as much as we can right now. I know people whoproduce factors that would support motor neurons,
have a "live for today" mentality, and I truly believeor perhaps the stem cells themselves might produce
that we should all follow that philosophy. NEVER turnsuch factors.
down a chance to have fun.What Do Existing Data Suggest?
My dilemma: How do I tell my friends and family? DoRecent data from Clement and colleagues show that
I wait for the condition to worsen, or get the wordin chimeric, genetically engineered mouse models,
out now and get the initial shock over with? I lookmotor neurons carry mutated SOD1 genes and glial
normal, so maybe now would be a better choicecells carry healthy genes. Survival is extended in
before I'm in a wheel chair or look sickly. I don't wantthese chimeric mice, as compared with nonchimeric
anyone to look at me differently. I don't want anymice in which all motor neurons and all glial cells carry
special attention and I certainly don't want anyone tomutated SOD1 genes. This finding suggests that if
feel sorry for me. (Thanks for those few who knewhealthy stem cells could get to the spinal cords of
about me earlier and didn't mention it.)patients with ALS, their survival might also be
One of my fears is that someone will get the idea toextended. It remains to be determined whether a
offer up a special intention for me at church...and I'llmechanism that compensates for a particular genetic
slowly sink into the pew. I appreciate the thoughtserror would apply to sporadic patients without that
and prayers, but please don't do that. I'll find a wayerror. Nevertheless, even if this form of therapy
to get you back...were effective only for patients with familial disease,
Seriously, my choice is to tell people as I see them orit would be a great leap forward.
make contact by email. Since I don't get out much,In previous experiments, intraspinal transplantation of
email seems the easiest way.neurons derived from a human teratoma cell line was
Now, on a more positive note, Dr. Baloh's Lab hasshown to ameliorate dysfunction and extend survival
made progress in converting skin stem cells intoin G93A SOD1 transgenic mice. Furthermore, the life
nerve cells in mice. I know I'm not a mouse, but it's aspan of G93A SOD1 mice was extended by
huge step. Once this science is further along, nerveintravenous administration of human umbilical cord
cells (from stem cells) can be utilized to replaceblood. The cells were shown to have migrated into
damaged nerve cells and hopefully find a cure forthe spinal cord and brain parenchyma and survived
ALS and many other neurological diseases.10-12 weeks after infusion. They exerted their
Just what is ALS? (Amyotrophic Lateral Sclerosis) is abeneficial effect even though only a low number of
motor neuron disease, first described in 1869 by thetransplanted cells expressed neural antigens. In
noted French neurologist Jean-Martin Charcot.another study, Sertoli cells, which are not neuronal
Although the cause is not completely understood, thestem cells, were implanted in the spinal cords of
last decade has brought a wealth of new scientificSOD1 transgenic mice and were shown to provide
understanding about the disease that provides hopetemporary protection to motor neurons in their
for the future.proximity. However, viable Sertoli cells were not
Lou Gehrig first brought national and internationalpresent at the time when the animals died.
attention to the disease in 1939 when he abruptlyPreliminary trials with autologous hematopoietic stem
retired from baseball after being diagnosed with ALS.cells have been reported in humans. In one, peripheral
Most commonly, the disease strikes people betweenblood-purified CD34+ cells were injected intrathecally
the ages of 40 and 70, and as many as 30,000into 3 patients with ALS. None reported side effects
Americans have the disease at any given time. Thisafter 6-12 months, but no clinical efficacy was
disease has cut short the lives of other such notablereported. In another, 7 patients received intraspinal
and courageous individuals as Hall of Fame pitcher Jimtransplantation of autologous bone marrow-derived
"Catfish" Hunter, Senator Jacob Javits, actors Michaelstem cells. Minor postoperative adverse events were
Zaslow and David Niven, creator of Sesame Streettransient, but muscle strength continued to decline.
Jon Stone, television producer Scott Brazil, boxingAfter 3 months, however, the investigators reported
champion Ezzard Charles, NBA Hall of Fame basketballa trend toward slowing of the decline in the proximal
player George Yardley, pro football player Glennmuscle groups of the lower limb in 4 patients and a
Montgomery, golfer Jeff Julian, golf caddie Brucemild increase in strength in 2 patients. Lack of placebo
Edwards, British soccer player Jimmy Johnstone,controls and longer follow-up preclude any inferences
musician Lead Belly (Huddie Ledbetter), photographerof efficacy from this study and none were made by
Eddie Adams, entertainer Dennis Day, jazz musicianthe investigators.
Charles Mingus, composer Dimitri Shostakovich,Stem Cell Research: Ethics, Economics, Policy, and
former vice president of the United States Henry A.Public Health
Wallace and U.S. Army General Maxwell Taylor.The ethics of performing human studies at this early
ALS is a neurodegenerative disease that usuallystage of stem cell research have been questioned,
attacks both upper and lower motor neurons andemphasizing the risks of premature human trials.
causes degeneration throughout the brain and spinalReports of stem cell transplantation performed in
cord. A common first symptom is a painlessChina without peer review of objective data on each
weakness in a hand, foot, arm or leg, which occurs inpatient before, immediately after, and at specific
more than half of all cases. Other early symptomslong-term points following the transplantation do not
include speech swallowing or walking difficulty. Theprovide sufficient scientific evidence to demonstrate
biological mechanisms that cause ALS are onlythat the treatment is safe and effective.
partially understood. The only known cause of ALS is"It is critical that scientists and clinicians are cautious,
a mutation of a specific gene: the SOD1 gene. Thisplan rigorous studies, and most importantly focus on
mutation is believed to make a defective protein thatkey laboratory experiments that will provide answers
is toxic to motor nerve cells. The SOD1 mutation,to the many challenges that still face this therapeutic
however, accounts for only 1 or 2 percent of ALSapproach," wrote Lucie Bruijn, PhD, the Science
cases, or 20 percent of the familial (inherited) cases.Director and Vice President of the ALS Association.
Familial ALS represents between five to 10 percent"For this therapy to be safe and have potential in the
of all cases. The rest arise spontaneously andclinic, it is critical that the appropriate studies are
mysteriously, making seemingly random attacks onconducted to learn more about the properties and
previously healthy adults. ALS can strike anyone,complexities of the various stem cells."
anytime.In response to limitations on the type of stem cell
Physicians have limited choices for treating ALS, andresearch that may be performed with federal funds,
the options that do exist have come into use withinthe American Academy of Neurology and the
the last 10 years. Studies suggest that patients'American Neurological Association -- the 2 leading
length of survival and quality of life are enhanced byprofessional neurology organizations in the United
night-time breathing assistance early in the course ofStates -- have both gone on record expressing the
the disease and by aggressive application of alternatebelief that both embryonic and adult stem cell
feeding options to assure good nutrition onceresearch should be pursued rigorously and under close
swallowing becomes difficult. At this time, riluzole isscrutiny, while respecting the concerns of their
the only drug that has been approved by the FDAmembers and the public, regarding important ethical
for treatment of ALS. In clinical trials, riluzole hasprinciples and values pertaining to research with
shown a slight benefit in modestly increasing survivalhuman embryonic stem cells.
time.The scientific concerns are 2-fold. First, because the
Stem cell and gene therapy are promising areas ofrealistic likelihood for success of any individual
research. In a variety of studies, ALS mouse modelsresearch effort is low, parallel research in multiple
are being used to develop treatments that maydirections is imperative for the field to advance
someday lead to similar human clinical trials. Generapidly. The essence of research is trial and error,
therapy is one field of research where The ALSwhich operates by identifying ineffective directions
Association is concentrating support for more study.and thereby focusing on those that hold promise. It is
If you would like to send a donation click here.usually a long time between initiating research and
More significant advances of research into ALS hasrealizing a successful treatment with clinical
occurred in the last decade than all of the time sinceapplications. Therefore, any delay in identification of a
Charcot identified the disease. Advances inpotentially effective therapeutic intervention
technology and the genetic revolution are aidingtranslates into delaying treatments for patients with
researchers in unlocking the ALS mystery. As morethe diseases in question. Second, excluding particular
scientists focus on this perplexing disease, thetypes of research from federal funding may translate
outlook for new understanding brightens each day.into an exclusion of this research from federal
What Are Stem Cells?oversight and protections, which might lead to its
Stem cells, also known as progenitor cells, are cellsmigration overseas. This may be detrimental to
that have not undergone differentiation to acquire aindividual patients and to the broader community of
specific structure or role; they have the potential topatients, clinicians, and scientists.
self-renew, divide, and differentiate into specializedIn November 2004, California citizens approved a
cell types. They are also sometimes termedreferendum measure to issue bonds to fund stem
"pluripotential" or "undifferentiated" cells because theycell research, including embryonic stem cell research
can differentiate and develop into various cell lines.at $300 million a year for 10 years. Since then,
The differentiation of stem cells into mature cells isseveral other states (Illinois, New Jersey, Maryland,
tightly regulated; otherwise, intricate plants andNew York, Delaware, and Wisconsin) are considering,
animals, with their many interrelated tissues, organs,or being asked to consider, initiatives for
and systems, could not exist.state-funded stem cell research to fill the federal
By contrast, mature or differentiated cells havefunding gap. This is motivated, in part, by the desire
acquired specific structures and roles, and in manyto remain on the forefront of medical research and
cases have lost the ability to divide and replicate. Alsoavert a brain drain toward states that provide an
in contrast to stem cells, malignant cells oreconomic environment more conducive to
"dedifferentiated" cells divide in an uncontrolledcutting-edge research. The ripple effect of the
fashion, and rather than resulting in useful,California initiative is expected to result in acceleration
differentiated, or specialized cells, these types of cellsof stem cell research nationwide.
threaten to kill the organism.Conclusion
Stem cell differentiation must be turned on, givenStem cell research carries promise for patients with
direction, and turned off as needed in order toALS and may result in the development of new
properly supply the basic building blocks of tissues intreatments to slow the progression of the disease.
different organ systems. This requirement for preciseThis hope needs to be tempered with caution
regulation applies to an even greater degree to thebecause of the early stages of stem cell research in
differentiation of neuronal progenitor cells, becausegeneral, and in ALS in particular, and because of the
effective neural function depends on establishingtrack record of the limited efficacy of all
precise linkages and interactions between differentpharmacologic interventions in transgenic murine and
individual neurons and classes of neurons.sporadic human ALS. Meticulous attention to the
By definition, stem cells, including neuronal progenitorethics and scientific rigor of future stem cell research
cells, are present in embryos. Stem cells may beshould be supported by clinicians, scientists, and
found in umbilical cord blood. In adults, these cells areparticipating patients alike.
present in bone marrow and in other organs in which